Comment
While I like membrane pacemaker damage theory of aging, there is issues such as neurons being able to live twice as long as animals they come from, and repeated cloning being possible both without signs of aging. There is also the claim telomere lengthening rejuvenates cell and tissue level phenotype from old to young.
I suspect that the damage is causing some internal clock or clocks to move and age us. Given again single cells like neurons can live twice as long as the animal and some believe can live more than ten times as long without genetic change.
If repeated cloning shows dividing cells can yield youthful tissue far beyond animal lifespan and same is true for nondividing cells the body seems able to handle damage fine bar some aging clock or program interfering.
